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September 16, 2024

Supplementary Data from A non-conserved histidine residue on KRAS drives paralog selectivity of the KRASG12D inhibitor MRTX1133

Miles A. Keats John J.W. Han Yeon‐Hwa Lee Chih-Shia Lee Ji Luo

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Keats et al. (2024) studied this question.

synapsesocial.com/papers/68e586c1b6db6435875234b4 https://doi.org/10.1158/0008-5472.27028360.v1

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Synapse has enriched 8 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Data from A non-conserved histidine residue on KRAS drives paralog selectivity of the KRASG12D inhibitor MRTX11332024
2Data from A non-conserved histidine residue on KRAS drives paralog selectivity of the KRASG12D inhibitor MRTX11332024
3Supplementary Figure S3 from The SOS1 Inhibitor MRTX0902 Blocks KRAS Activation and Demonstrates Antitumor Activity in Cancers Dependent on KRAS Nucleotide Loading2024
4Supplementary Figure S1 from The SOS1 Inhibitor MRTX0902 Blocks KRAS Activation and Demonstrates Antitumor Activity in Cancers Dependent on KRAS Nucleotide Loading2024
5Supplementary Figure S5 from The SOS1 Inhibitor MRTX0902 Blocks KRAS Activation and Demonstrates Antitumor Activity in Cancers Dependent on KRAS Nucleotide Loading2024
6Supplementary Figure S2 from The SOS1 Inhibitor MRTX0902 Blocks KRAS Activation and Demonstrates Antitumor Activity in Cancers Dependent on KRAS Nucleotide Loading2024
7Supplementary Figure S4 from The SOS1 Inhibitor MRTX0902 Blocks KRAS Activation and Demonstrates Antitumor Activity in Cancers Dependent on KRAS Nucleotide Loading2024
8Supplementary Figure S7 from The SOS1 Inhibitor MRTX0902 Blocks KRAS Activation and Demonstrates Antitumor Activity in Cancers Dependent on KRAS Nucleotide Loading2024