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September 16, 2024

Supplementary Data from A non-conserved histidine residue on KRAS drives paralog selectivity of the KRASG12D inhibitor MRTX1133

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Keats et al. (2024) studied this question.

synapsesocial.com/papers/68e586b5b6db6435875231d7 https://doi.org/10.1158/0008-5472.27028360

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Data from A non-conserved histidine residue on KRAS drives paralog selectivity of the KRASG12D inhibitor MRTX11332024
2Data from A non-conserved histidine residue on KRAS drives paralog selectivity of the KRASG12D inhibitor MRTX11332024
3Supplementary Figure S3 from The SOS1 Inhibitor MRTX0902 Blocks KRAS Activation and Demonstrates Antitumor Activity in Cancers Dependent on KRAS Nucleotide Loading2024
4Supplementary Figure S1 from The SOS1 Inhibitor MRTX0902 Blocks KRAS Activation and Demonstrates Antitumor Activity in Cancers Dependent on KRAS Nucleotide Loading2024
5Supplementary Figure S5 from The SOS1 Inhibitor MRTX0902 Blocks KRAS Activation and Demonstrates Antitumor Activity in Cancers Dependent on KRAS Nucleotide Loading2024