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August 23, 2024Frontiers in Bioscience-LandmarkOpen Access

Inhibiting the NF-κB/DRP1 Axis Affords Neuroprotection after Spinal Cord Injury via Inhibiting Polarization of Pro-Inflammatory Microglia

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Authors

SCSong ChenKZKaihui ZhangCLCheng Luo

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Chen et al. (2024) studied this question.

synapsesocial.com/papers/68e5b28db6db64358754c028https://doi.org/10.31083/j.fbl2908307
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Nfkbia-driven neuroinflammatory pathways mediate depression following spinal cord injury2025
  2. 2MSR405: Inhibiting Neuroinflammation after Spinal Cord Injury in Rats2024 · 6 citations
  3. 3Role of nuclear factor-κB (NFκB) in microglial polarization in correlation with neuroinflammatory mechanism at the hippocampal cornu ammonis (CA) 1 region after acute and chronic phase of global ischemic brain injury in rats2025
  4. 4SP1 transcriptionally activates HTR2B to aggravate traumatic spinal cord injury by shifting microglial M1/M2 polarization2024 · 17 citations
  5. 5Daphnoretin inhibited SCI-induced inflammation and activation of NF-κB pathway in spinal dorsal horn2024