The presence of low voltage areas (LVA) and/or scars in the left atrial anterior wall is a significant arrhythmogenic substrate for atrial fibrillation (AF) and atrial tachycardia (AT) 1,2. The anterior mitral line (AML) is one of the effective methods for modifying and treating AF and AT 3,4; however, this linear ablation can occasionally create iatrogenic reentrant substrates for complex AT 5. Performing a linear ablation at the mitral isthmus (MI) following AML carries a substantial risk of electrical isolation of the left atrial appendage (LAA).
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Kimura et al. (2024) studied this question.
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