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July 11, 2024Arteriosclerosis Thrombosis and Vascular BiologyOpen Access

Decoding Endothelial MPL and JAK2V617F Mutation: Insight Into Cardiovascular Dysfunction in Myeloproliferative Neoplasms

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Authors

HZHaotian ZhangNKNicholas KafeitiKMKyla Masarik

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Cite This Study

Zhang et al. (2024) studied this question.

synapsesocial.com/papers/68e609c9b6db64358759d2a8https://doi.org/10.1161/atvbaha.124.321008
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1JAK2V617F-positive clonal hematopoiesis in germline BRCA1 versus BRCA2 mutation carriers2026
  2. 2JAK2 V617F Mutations and Thrombosis in BCR-ABL1 Negative Myeloproliferative Neoplasms: Clinical Profile and Hematologic Insights2026
  3. 3Association between JAK2V617F variable allele frequency and risk of thrombotic events in patients with myeloproliferative neoplasms2024 · 8 citations
  4. 4The anti-MPL antibody AMM2 prevents cardiovascular dysfunction in a mouse model of myeloproliferative neoplasms2025
  5. 5HiJAKing the Hematopoietic System: A Low-Frequency JAK2V617F Clone Drives Myeloproliferative Neoplasm Pathology2025 · 3 citations