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July 1, 2024Journal of Experimental & Clinical Cancer ResearchOpen Access

LILRB4 on multiple myeloma cells promotes bone lesion by p-SHP2/NF-κB/RELT signal pathway

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Authors

HWHongying WangLWLei WangHLHuiwen Luan

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Cite This Study

Wang et al. (2024) studied this question.

synapsesocial.com/papers/68e61ca0b6db6435875aee09https://doi.org/10.1186/s13046-024-03110-y
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1LILRB4 regulates multiple myeloma development through STAT3-PFKFB1 pathway2024 · 12 citations
  2. 2LILRB4 regulates multiple myeloma development through STAT3-PFKFB1 pathway2024
  3. 3LILRB4 represents a promising target for immunotherapy by dual targeting tumor cells and myeloid-derived suppressive cells in multiple myeloma2024 · 12 citations
  4. 4LILRB4 drives proinflammatory cytokine secretion in multiple myeloma to promote the immune-cell dysfunction via the generation of myeloid-derived suppressive cells2025
  5. 5Abstract 364: Targeting LILRB1 to sensitize human myeloma to ferroptosis through disrupting cholesterol homeostasis2024