Eisenmenger syndrome¹ is a constellation of symptoms that arise from congenital heart defects. It occurs when a long standing left to right anatomic cardiac shunt caused by congenital heart defect (typically VSD and ASD and less likely PDA) causes pulmonary hypertension and eventually reversal of shunt into a cyanotic right to left shunt, manifesting in early life. This case discusses the clinical presentation, diagnostic workup, and management of a patient of Eisenmenger syndrome secondary to an uncorrected ventricular septal defect (VSD) that presented with Acute Dengue Fever. A 22 year old female presented to the emergency department with history of self induced abortion at home and chief complaints of fever with chills, dyspnea on exertion NYHA Grade 3, giddiness and ghabraman. Evaluation of this patient revealed hypoxemia, central cyanosis, clubbing, cardiomegaly and VSD with a bi-directional shunt and moderate PAH. Patient was given oxygen support (initially for stabilization), medical care², blood products, eventually anticoagulation, and finally, referred for surgical care³.
No takes yet. Share an insight, caveat, or question.
Jain et al. (2024) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: