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April 9, 2024Neurology

The Relationship Between GSK3Beta Activity and TDP-43 Mediated Neuritis in Neurodegenerative Diseases (P6-9.005)

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Abstract

We want to see if regulating GSK3β activity could weaken cystic TDP-43 intermediate neuroinflammatory in degenerative diseases.

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Cite This Study

Eu Jene Choi (2024) studied this question.

synapsesocial.com/papers/68e6fdadb6db643587677fee https://doi.org/10.1212/wnl.0000000000204393

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Inhibiting Glycogen Synthase Kinase 3 Suppresses TDP-43-Mediated Neurotoxicity in a Caspase-Dependent MannerOriginal Research · 2026 · 1 citations
2TDP-43 pathology triggers neuroinflammation and cognitive impairment by inducing microglial necroptosisOriginal Research · 2026 · 1 citations
3Role of GSK-3 Inhibition in Alzheimer’s Disease Therapy2025 · 2 citations
4TDP-43 phosphorylation: Exploring kinases, phosphatases, and therapeutic potential in neurodegeneration.Review · 2026
5The dual role of glycogen synthase kinase-3 beta (GSK3β) in neurodegenerative pathologies: interplay between autophagy and disease progression2025

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