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March 1, 2024Open Access

Figure S1 from TNFRSF19 within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling

Xianglin Zuo Xuchun Wang Tingzheng Ma Shuhan Chen Pingping Cao He Cheng Nan Yang Xiao Han Wei Gao Xiaoyu Liu Yujie Sun

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Abstract

The TNFRSF19 within 13q12.12 risk chromatin region functions as a lung cancer suppressor by binding Wnt3a to inhibit Wnt/β-catenin signaling.

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Cite This Study

Zuo et al. (2024) studied this question.

synapsesocial.com/papers/68e76734b6db6435876dcb64 https://doi.org/10.1158/1541-7786.25325031.v1

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Figure S5 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024
2Table S1 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024
3Table S1 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024
4Figure S2 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024
5Table S2 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024