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March 1, 2024Open Access

Figure S5 from TNFRSF19 within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling

Xianglin Zuo Xuchun Wang Tingzheng Ma Shuhan Chen Pingping Cao He Cheng Nan Yang Xiao Han Wei Gao Xiaoyu Liu Yujie Sun

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Abstract

Analysis of the TNFRSF19 gene promoter methylation based on TCGA and GEO database.

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Cite This Study

Zuo et al. (2024) studied this question.

synapsesocial.com/papers/68e7681cb6db6435876dd0a7 https://doi.org/10.1158/1541-7786.25325019

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Figure S1 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024
2Figure S3 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024
3Table S1 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024
4Table S1 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024
5Figure S2 from <i>TNFRSF19</i> within the 13q12.12 Risk Locus Functions as a Lung Cancer Suppressor by Binding Wnt3a to Inhibit Wnt/β-Catenin Signaling2024