Targeting Alzheimer's pathophysiology with carbon dots: from mechanisms to therapy

Shourya Tripathi; Ramandeep Singh; Srishty Jaiswal; Mitali Sethi; Rafquat Rana; Keerti Mishra; Manish K. Chourasia

doi:10.1039/d5tb02134k

December 8, 2025Journal of Materials Chemistry B

Targeting Alzheimer's pathophysiology with carbon dots: from mechanisms to therapy

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Key Points

Carbon dots reduce oxidative stress, targeting pathophysiology in Alzheimer's disease and offering therapeutic potential.
The therapy involves disrupting Aβ and tau aggregation while crossing the blood–brain barrier.
Assessment of carbon dots demonstrates effectiveness in tackling Alzheimer's mechanisms and fostering treatment innovation.
The findings suggest that carbon dots may lead to new approaches in Alzheimer's therapy with implications for future research.

Implication

Therapy using carbon dots mitigates oxidative stress in Alzheimer's, indicating potential for novel treatment pathways.

Abstract

Carbon dots cross the blood–brain barrier, disrupt Aβ and tau aggregation, and mitigate oxidative stress, offering a multifunctional nanoplatform for Alzheimer's disease therapy.

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Cite This Study

Tripathi et al. (2025) studied this question.

synapsesocial.com/papers/69401f062d562116f28f9e4f https://doi.org/10.1039/d5tb02134k

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Targeting Alzheimer's pathophysiology with carbon dots: from mechanisms to therapy

Key Points

Implication

Abstract

Discussion

Cite This Study

Targeting Alzheimer's pathophysiology with carbon dots: from mechanisms to therapy

Key Points

Implication

Abstract

Discussion

Cite This Study

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