Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
November 25, 2025Scientific ReportsOpen Access

The slow delayed rectifier K+ current is differently regulated under baseline conditions and following β-adrenergic stimulation in canine ventricular cardiomyocytes

View Full Paper
Ask AI
Bookmark
Share

Authors

BHBalázs HorváthUniversity of DebrecenZKZsigmond Máté KovácsUniversity of DebrecenCDCsaba DienesUniversity of Debrecen

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Horváth et al. (2025) studied this question.

synapsesocial.com/papers/6940379e2d562116f290a2f6https://doi.org/10.1038/s41598-025-25754-2
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Effects of sustained β-adrenergic stimulation on ionic currents of cultured adult guinea pig cardiomyocytes2002 · 48 citations
  2. 2Epac-mediated Activation of Phospholipase Cɛ Plays a Critical Role in β-Adrenergic Receptor-dependent Enhancement of Ca2+ Mobilization in Cardiac Myocytes2006 · 187 citations
  3. 3Calmodulin kinase II inhibition limits the pro-arrhythmic Ca2+waves induced by cAMP-phosphodiesterase inhibitors2016 · 37 citations
  4. 4Epac and Phospholipase Cϵ Regulate Ca2+ Release in the Heart by Activation of Protein Kinase Cϵ and Calcium-Calmodulin Kinase II2008 · 209 citations
  5. 5Inhibition of forskolin-induced neurite outgrowth and protein phosphorylation by a newly synthesized selective inhibitor of cyclic AMP-dependent protein kinase, N-[2-(p-bromocinnamylamino)ethyl]-5-isoquinolinesulfonamide (H-89), of PC12D pheochromocytoma cells.1990 · 1,501 citations