Left ventricular damage and dysfunction can begin in early stages of chronic kidney disease when eGFR declines to approximately 60 ml/min/1.73 m².
Myocardial damage and dysfunction start in early stages of CKD, prompting the reclassification of 'uraemic cardiomyopathy' to 'CKD-associated cardiomyopathy' to encourage earlier recognition and preventive therapies.
Absolute Event Rate: 0% vs 0%
Uraemic cardiomyopathy is a term widely used to describe the severe myocardial disease characterized by left ventricular hypertrophy and diffuse interstitial fibrosis that occurs in kidney failure. The causative factors are multiple and include many of the haemodynamic and metabolic abnormalities that are present in patients with chronic kidney disease (CKD). These abnormalities start to cause left ventricular damage and dysfunction in the early stages of CKD, probably from an estimated glomerular filtration rate of approximately 60 ml/min/1.73 m
Edwards et al. (Fri,) reported a other. Left ventricular damage and dysfunction can begin in early stages of chronic kidney disease when eGFR declines to approximately 60 ml/min/1.73 m².
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