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October 16, 2007Nucleic Acids ResearchOpen Access

Heme oxygenase-1 induction by NRF2 requires inactivation of the transcriptional repressor BACH1

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Authors

JRJohn F. ReichardCincinnati Health DepartmentGMGregory T. MotzVeterans Health AdministrationAPAlvaro PugaUniversity of Cincinnati Medical Center

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Cite This Study

Reichard et al. (2007) studied this question.

synapsesocial.com/papers/697fabb652db303aecf982b5https://doi.org/10.1093/nar/gkm638
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Bach Proteins Belong to a Novel Family of BTB-Basic Leucine Zipper Transcription Factors That Interact with MafK and Regulate Transcription through the NF-E2 Site1996 · 670 citations
  2. 2Monomeric and Dimeric bZIP Transcription Factor GCN4 Bind at the Same Rate to Their Target DNA Site2003 · 56 citations
  3. 3Normal keratinization in a spontaneously immortalized aneuploid human keratinocyte cell line.1988 · 4,203 citations
  4. 4Thermodynamics of the As(III)−Thiol Interaction: Arsenite and Monomethylarsenite Complexes with Glutathione, Dihydrolipoic Acid, and Other Thiol Ligands2005 · 206 citations
  5. 5Bach1 Competes with Nrf2 Leading to Negative Regulation of the Antioxidant Response Element (ARE)-mediated NAD(P)H:Quinone Oxidoreductase 1 Gene Expression and Induction in Response to Antioxidants2005 · 415 citations