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LINC00312 demonstrates potential to regulate inflammation and apoptosis in chondrocytes, suggesting novel OA treatment.
LINC00312 alleviates IL-1β-induced chondrocyte inflammation and apoptosis by acting as a ceRNA to regulate the miR-331-3p/DUSP5 axis. This suggests that LINC00312 may serve as a novel therapeutic target for OA.
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Zhao et al. (2026) studied this question.
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