Analysis reveals ETV4-STAT6 activation inhibits ferroptosis in thyroid cancer, suggesting potential therapeutic strategies.
ETV4 transcriptionally activated STAT6 to inhibit ferroptosis and promote immune escape in THCA. Targeting the ETV4-STAT6 axis may offer a novel therapeutic strategy, potentially inhibiting ferroptosis and enhancing immune response in THCA patients.
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Zong et al. (2026) studied this question.
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