Why the study?
Does the extent of autonomic activation following cerebral ischemia differ between hypertensive and normotensive humans?
Does the extent of autonomic activation following cerebral ischemia differ between hypertensive and normotensive humans?
The findings suggest the insular cortex may play a role in the pathogenesis of essential hypertension based on differences in autonomic activation post-stroke.
No immediate clinical implications post-stroke; hypothesis-generating for insular role in hypertension.
Our findings suggest a difference in cortical control of autonomic function between hypertensive and normotensive patients after stroke and point to a possible role of the insular cortex in the pathogenesis of essential hypertension.
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Sander et al. (1996) studied this question.
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