Lack of a Tricuspid Regurgitation Doppler Signal and Pulmonary Hypertension by Invasive Measurement

Figures

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  Figure 1. Flow diagram of the study population. mPAP indicates mean pulmonary artery pressure; RHC, right heart catheterization; TRV, tricuspid regurgitation velocity; TTE, transthoracic echocardiogram.

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  Figure 2. Distribution of mean pulmonary pressures in those with and without tricuspid regurgitant velocity (TRV) on echocardiogram. Histogram of the distribution of invasive pulmonary pressures stratified by presence or absence of a reported TRV.

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  Figure 3. Echocardiographic and clinical features associated with pulmonary hypertension among subjects without a reported tricuspid regurgitant velocity. Multivariate regression analysis of (A) echocardiographic and (B) clinical variables selected a priori based on clinical knowledge. Pulmonary hypertension was associated with left atrial enlargement, right ventricular dysfunction, body mass index, elevated brain natriuretic peptide, and prevalent heart failure. RV dilation and dysfunction were analyzed as binary variables. Any degree of dilation or dysfunction was compared with absence of dilation or dysfunction. Odds ratio for continuous variables represents the difference between the 25th and 75th percentiles. BNP indicates brain natriuretic peptide; CKD, chronic kidney disease; COPD, chronic obstructive pulmonary disease; LA, left atrium; PH, pulmonary hypertension; RV, right ventricle.