Why the study?
How do changes in breathing interval and tidal volume affect respiratory sinus arrhythmia and vagal cardiac outflow in healthy humans?
How do changes in breathing interval and tidal volume affect respiratory sinus arrhythmia and vagal cardiac outflow in healthy humans?
Measurements of respiratory sinus arrhythmia during controlled breathing suggest that phasic respiration-related changes of vagal motoneuron activity begin in expiration and are incompletely expressed at fast breathing rates.
No takes yet. Share an insight, caveat, or question.
RSA measures require breathing-rate standardization in practice; leaves open vagal dynamics in disease.
D. L. Eckberg (1983) studied this question.
Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context: