Stimulation of baroreceptors in the right subclavian artery decreases sympathetic and increases parasympathetic (vagal) efferent nerve activity in animal models.
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Baroreceptor stimulation in the subclavian artery modulates autonomic outflow in animals; leaves open translation to human cardiovascular regulation.
The effect of stimulation of the baroreceptors of the right subclavian artery upon the efferent impulse activity of the cardioregulatory and abdominal sympathetic nerves was investigated in decerebrate cats and anesthetized dogs. Increase in the pressure in the isolated and perfused right subclavian-carotid arterial segment diminished or abolished the impulse activity in the cardiac and abdominal sympathetic nerves. Simultaneously there was an increase of impulse activity in the cardiac vagus. No appreciable change of impulse activity in the long ciliary nerve was noticed. Impulse activity in the cardiac vagus nerve was found to be predominant during expiration both in decerebrated and anesthetized dogs with bilateral occlusion of the common carotid arteries.
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Hiromasa Okada (1964) studied this question.
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