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January 1, 1987Free Radical Biology and MedicineOpen Access

Protein oxidation and loss of protease activity may lead to cataract formation in the aged lens

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Authors

ATAllen TaylorMedStar Heart & Vascular Institute
Kelvin J.A. Davies
Kelvin J.A. DaviesBrighton and Sussex Medical School

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Taylor et al. (1987) studied this question.

synapsesocial.com/papers/69d98a200d540cafc583616bhttps://doi.org/10.1016/0891-5849(87)90015-3
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Glutathione peroxidase, glutathione reductase and superoxide dismutase in the aging lens1984 · 64 citations
  2. 2Formation of a 55,000 molecular-weight crosslinked .beta. crystallin dimer in the calcium treated lens. A model for cataract1985 · 43 citations
  3. 3The eye lens has an active ubiquitin-protein conjugation system.1986 · 93 citations
  4. 4Protein damage and degradation by oxygen radicals. I. general aspects.1987 · 1,640 citations
  5. 5Proteins damaged by oxygen radicals are rapidly degraded in extracts of red blood cells.1987 · 340 citations