High levels of beta(2)AR overexpression in mice lead to cardiomyopathy and heart failure, though requiring higher expression and having a slower onset than beta(1)AR overexpression.
High-level beta(2)AR overexpression induces cardiomyopathy in mice; extends betaAR isoform data but leaves clinical translation open.
A high level of beta(2)AR overexpression results in cardiomyopathy and heart failure. The onset was slower and the expression levels of receptors required are much higher than previously described for the beta(1)AR overexpression.
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X. Du (2000) studied this question.
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