End-stage heart failure is associated with a specific compensatory upregulation of catalase gene expression in response to oxidative stress, without changes in SOD or GPX.
Highlights selective catalase upregulation in end-stage HF; hypothesis-generating and should not yet change practice.
Increased oxidative stress in human end-stage heart failure may result in a specific upregulation of catalase gene expression as a compensatory mechanism, whereas SOD and GPX gene expression remain unaffected.
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Dieterich et al. (2000) studied this question.
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