In vitro analysis reveals RPE cells facilitate macrophage migration during T. gondii infection, suggesting new treatment avenues.
RPE cells are not passive targets but active participants in the ocular immune response to T. gondii. By secreting IL-6 and IL-10, they establish a chemotactic environment that recruits macrophages. These insights identify the RPE-cytokine-macrophage axis as a potential therapeutic target in ocular toxoplasmosis.
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Násare et al. (2026) studied this question.
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