A theoretical computational model supports the hypothesis that ATP release by red blood cells and upstream conducted responses are key mechanisms in metabolic blood flow regulation.
No takes yet. Share an insight, caveat, or question.
Supports microvascular regulation models via ATP release; leaves open human validation before any clinical consideration.
Arciero et al. (2008) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: