Key result
High-density mapping of adenosine-sensitive atrial tachycardia visualized dull components along the tricuspid annulus representing the slow conduction zone, which was successfully ablated without recurrence at 20 months.
Why the study?
The underlying mechanism of adenosine-sensitive atrial tachycardia originating from the atrioventricular annulus remains unclear, and identifying the entire reentrant circuit is challenging.
Case Report (n=1)
No
Answers are grounded in this paper and cite their sources.
High-density mapping can visualize dull components along the tricuspid annulus that represent the slow conduction zone in adenosine-sensitive atrial tachycardia, aiding in successful ablation.
The underlying mechanism of adenosine-sensitive atrial tachycardia (AT) originating from the atrioventricular annulus remains unclear. Adenosine-sensitive AT represents triggered activity or automaticity based on centrifugal patterns in activation maps and termination or suppression by adenosine triphosphate. 1 Recently, some reports have suggested that the mechanism is re-entry with a slow conduction zone (SCZ) composed of calcium channel-dependent tissues because adenosine-sensitive AT is sensitive to adenosine triphosphate and verapamil and can be terminated and initiated by atrial programmed pacing.
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Maeda et al. (2026) conducted a case report in Adenosine-sensitive atrial tachycardia (n=1). High-density mapping and radiofrequency catheter ablation was evaluated on Termination of tachycardia and freedom from recurrence. High-density mapping of adenosine-sensitive atrial tachycardia visualized dull components along the tricuspid annulus representing the slow conduction zone, which was successfully ablated without recurrence at 20 months.
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