Synapse
⌘+K
Synapse
PulseExploreJournal ClubResearchersJournals
Instagram
HomeJournal ClubExplore
April 9, 2005European Journal of Heart Failure

Pulmonary Vascular Remodeling in Pulmonary Hypertension Due to Chronic Heart Failure

View Full Paper
Ask AI
Bookmark
Share

Population

17 heart transplant recipients with preoperative chronic heart failure who died shortly after heart…

Design

Cohort

Authors

JDJuan F. DelgadoECEsther CondeVSVioleta Sánchez

Discussion

Loading...

Member takes

Overview

Greater medial thickness in at-risk PHT may flag post-transplant RV failure risk; hypothesis-generating for vascular remodeling in chronic HF.

Key Points

  • This study aims to investigate pulmonary vascular changes in patients with chronic heart failure and their correlation with haemodynamic data.
  • Studied 17 heart transplant recipients with preoperative chronic heart failure who died shortly after transplantation.
  • Obtained preoperative haemodynamic information immediately before heart transplantation.
  • Assessed vascular lesions in muscular arteries using linear morphometry.
  • Mean transpulmonary gradient was 8.9±4.5 mm Hg with pulmonary vascular resistance at 2.25±1.34 Wu.
  • Six patients exhibited at-risk pulmonary hypertension based on the threshold criteria (TPG>12 mm Hg or PVR>2.5 Wu).
  • Medial thickness was significantly higher in patients with at-risk pulmonary hypertension (23.82±7.23%) compared to those without (17.16±3.24%, p=0.018).

Structured PICO

P
Population
17 heart transplant recipients with preoperative chronic heart failure who died shortly after heart transplantation
O
Outcome
Vascular lesions in muscular arteries assessed by linear morphometry (medial thickness) and correlation with preoperative haemodynamic datasurrogate

Medial hypertrophy of muscular pulmonary arteries is common in patients with chronic heart failure, even without at-risk pulmonary hypertension, potentially explaining post-transplant right ventricular failure risk.

Cite This Study

Delgado et al. (2005) studied this question.

synapsesocial.com/papers/6a064cab0924dc877016d8cehttps://doi.org/10.1016/j.ejheart.2004.10.021
View Full Paper
Ask AI
Bookmark
Share