Key result
Aerobic endurance training in rats with myocardial infarction reduced left and right ventricular weights by 15% and 23%, and myocyte length and width by 12% and 20% versus sedentary controls.
Why the study?
Does aerobic endurance training improve cardiomyocyte hypertrophy, contractility, and Ca2+ handling in a rat model of myocardial infarction?
Population
36 adult female Sprague-Dawley rats, 4 weeks after ligation of the left coronary artery or sham operation
Comparison
Aerobic endurance training on a treadmill vs Sedentary controls (sham-operated and infarcted)
Design
Preclinical
Follow-up
8 weeks
Authors
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Caution against clinical adoption post-MI; leaves open translation of hypertrophy attenuation from this rat model.
Does aerobic endurance training improve cardiomyocyte hypertrophy, contractility, and Ca2+ handling in a rat model of myocardial infarction?
Aerobic endurance training attenuates ventricular and cellular hypertrophy and restores contractile function and Ca2+ handling in a rat model of myocardial infarction.
Ulrik Wisløff (2002) studied Myocardial infarction (n=36). Aerobic endurance training vs. Sedentary controls was evaluated on Myocardial mass, myocyte dimensions, contractile function, Ca2+ handling, and myofilament responsiveness to Ca2+. Aerobic endurance training in rats with myocardial infarction reduced left and right ventricular weights by 15% and 23%, and myocyte length and width by 12% and 20% versus sedentary controls.
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