Key result
Internodal conduction time was prolonged by an average of 37 msec in patients with an endocardial cushion defect compared to other groups, explaining their prolonged P-R interval.
Why the study?
Is the prolonged P-R interval in patients with endocardial cushion defects caused by prolonged internodal conduction time or A-V nodal-His-Purkinje conduction delay?
Population
30 patients undergoing open heart surgery
Comparison
Intraoperative measurement of conduction time… vs Patients with secundum type ASD, sinus venosus…
Design
Cross-sectional
Authors
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Internodal delay explains PR prolongation in endocardial cushion defects; hypothesis-generating and should not yet change practice.
Observational (n=30)
Is the prolonged P-R interval in patients with endocardial cushion defects caused by prolonged internodal conduction time or A-V nodal-His-Purkinje conduction delay?
Effect estimate: 37 msec prolongation
The prolonged P-R interval often seen in patients with endocardial cushion defects is due to prolonged internodal conduction time rather than abnormal A-V nodal-His-Purkinje conduction.
Waldo et al. (1973) conducted an observational in Endocardial cushion defect (n=30). Conduction time measurement during open heart surgery vs. Patients with secundum ASD, sinus venosus ASD, or intact atrial septum was evaluated on Internodal conduction time (37 msec prolongation). Internodal conduction time was prolonged by an average of 37 msec in patients with an endocardial cushion defect compared to other groups, explaining their prolonged P-R interval.
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