Key result
This review summarizes the mechanisms by which HIV-1 alternative RNA splicing is regulated by viral cis-acting elements and cellular RNA binding proteins.
Population
HIV-1 infected cells, human T cells, human macrophages, and rodent cells
Design
Review
Authors
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No immediate clinical application; leaves open targeted antiviral development based on splicing mechanisms.
This review summarizes the mechanisms of HIV-1 alternative RNA splicing regulation by viral cis-acting elements and cellular RNA binding proteins.
Stoltzfus et al. (2005) conducted a review in HIV-1 infection. This review summarizes the mechanisms by which HIV-1 alternative RNA splicing is regulated by viral cis-acting elements and cellular RNA binding proteins.
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