Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
December 1, 1997European Journal of Immunology

Myelin oligodendrocyte glycoprotein‐induced autoimmune encephalomyelitis is chronic/relapsing in perforin knockout mice, but monophasic in Fas‐ and Fas ligand‐deficient lpr and gld mice

View Full Paper
Ask AI
Bookmark
Share

Authors

UMUrsula MalipieroUniversity Hospital of ZurichKFKarl FreiUniversity of California, RiversideKSKatharina‐Susanne SpanausUniversity Hospital of Zurich

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Malipiero et al. (1997) studied this question.

synapsesocial.com/papers/6a0fcf66d8c5cf602efd30b2https://doi.org/10.1002/eji.1830271211
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Differentiation of cerebellar bipotential glial preecurosrs into oligodendrocytes in primary culture: Development profile of surface antigens and mitotic activity1987 · 140 citations
  2. 2Antigen presentation and tumor cytotoxicity by interferon‐γ‐treated microglial cells1987 · 644 citations
  3. 3Human autoreactive CD4+ T cell clones use perforin- or Fas/Fas ligand-mediated pathways for target cell lysis1997 · 100 citations
  4. 4IFN-gamma plays a critical down-regulatory role in the induction and effector phase of myelin oligodendrocyte glycoprotein-induced autoimmune encephalomyelitis1996 · 638 citations
  5. 5Tumor Necrosis Factor α and Lymphotoxin α Are Not Required for Induction of Acute Experimental Autoimmune Encephalomyelitis1997 · 198 citations