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April 6, 1999CirculationOpen Access

Inhibition of p38 Mitogen-Activated Protein Kinase Decreases Cardiomyocyte Apoptosis and Improves Cardiac Function After Myocardial Ischemia and Reperfusion

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Authors

XLXin L.Nanyang Technological UniversitySKSanjay KumarGlaxoSmithKline (United States)FGFeng GaoGenVec

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Cite This Study

L. et al. (1999) studied this question.

synapsesocial.com/papers/6a0fdcb0d13714ec96feb653https://doi.org/10.1161/01.cir.99.13.1685
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Reperfusion injury induces apoptosis in rabbit cardiomyocytes.1994 · 1,445 citations
  2. 2Possible Involvement of Stress-Activated Protein Kinase Signaling Pathway and Fas Receptor Expression in Prevention of Ischemia/Reperfusion-Induced Cardiomyocyte Apoptosis by Carvedilol1998 · 237 citations
  3. 3Tissue-specific Pattern of Stress Kinase Activation in Ischemic/Reperfused Heart and Kidney1997 · 367 citations
  4. 4Apoptosis Induced by Withdrawal of Trophic Factors Is Mediated by p38 Mitogen-activated Protein Kinase1997 · 471 citations
  5. 5Apoptosis in Human Acute Myocardial Infarction1997 · 826 citations