Key result
Adenosine administration terminated 100% of sustained focal annular ventricular and atrial tachycardias, providing evidence for a shared mechanism of intracellular calcium overload.
Why the study?
Do focal annular atrial and ventricular tachycardias share a common mechanism characterized by adenosine sensitivity?
Population
49 consecutive patients with sustained, focal annular tachycardia originating from the peri-tricuspid and…
Design
Cohort
Authors
Loading...
Supports adenosine testing to identify focal annular tachycardias; leaves open prospective confirmation of shared calcium-overload mechanism.
Observational (n=49)
Do focal annular atrial and ventricular tachycardias share a common mechanism characterized by adenosine sensitivity?
Peri-annular atrial and ventricular tachycardias share a common mechanism of adenosine sensitivity, consistent with intracellular calcium overload and triggered activity.
Ip et al. (2014) conducted an observational in Sustained focal annular tachycardia (n=49). Adenosine administration during electrophysiological evaluation was evaluated on Tachycardia termination in response to adenosine. Adenosine administration terminated 100% of sustained focal annular ventricular and atrial tachycardias, providing evidence for a shared mechanism of intracellular calcium overload.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: