Why the study?
Do the conduction properties of the crista terminalis differ in AF patients who develop typical atrial flutter due to amiodarone therapy compared to those who do not?
Do the conduction properties of the crista terminalis differ in AF patients who develop typical atrial flutter due to amiodarone therapy compared to those who do not?
Patients with AF who develop typical atrial flutter on amiodarone exhibit a greater predisposition to transverse conduction delay and block in the crista terminalis.
No takes yet. Share an insight, caveat, or question.
Should not yet change amiodarone use in AF; supports crista terminalis delay hypothesis for drug-induced flutter.
TAI et al. (2003) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: