Key result
Arterial alterations in end-stage renal disease include large artery remodeling and stiffening, driven by multifactorial causes including extensive arterial calcifications.
Population
Patients with end-stage renal disease (ESRD)
Design
Review
Authors
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Nonatheromatous remodeling and stiffening should factor into ESRD CV risk assessment; extends atherosclerosis-centric models for mechanistic research.
Cardiovascular morbidity in ESRD is driven not only by atherosclerosis but also by nonatheromatous arterial remodeling and stiffening.
G. London (2002) conducted a review in End-stage renal disease (ESRD). Arterial alterations in end-stage renal disease include large artery remodeling and stiffening, driven by multifactorial causes including extensive arterial calcifications.
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