Key result
Prolonged pMCS in cardiogenic shock may trigger a pro-inflammatory response leading to vasodilation.
Why the study?
In cardiogenic shock, initial vasoconstriction may transition over time to vasodilation, which is often triggered by percutaneous mechanical circulatory support.
This review elucidates the hemodynamics and factors leading to vasodilation in patients with cardiogenic shock undergoing prolonged percutaneous mechanical circulatory support.
Answers are grounded in this paper and cite their sources.
Suggests monitoring for vasodilatory shock in prolonged MCS; leaves open prospective validation of inflammatory mechanisms.
Hemodynamics play an important role in cardiogenic shock assessment for prognosis estimation and for phenotyping cardiogenic shock. This is best done by pulmonary artery catheters. In general, at the beginning of cardiogenic shock, patients have vasoconstriction, which over time may lead to vasodilation. This is often triggered by percutaneous mechanical circulatory support. This review will elucidate the hemodynamics and the factors that possibly lead to vasodilation in patients with mechanical circulatory support.
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Hölger Thiele (2022) conducted a review in Cardiogenic shock. Percutaneous mechanical circulatory support was evaluated. Prolonged percutaneous mechanical circulatory support in patients with cardiogenic shock may trigger a pro-inflammatory response that leads to vasodilation over time.
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