Key result
End-tidal carbon dioxide cut-off of 30 mmHg predicts MACE risk in ACS patients.
Why the study?
The study was conducted to examine the capacity of EtCO2 levels to predict MACE in patients with acute coronary syndrome and evaluate the relationship between EtCO2 values and established risk scoring systems (TIMI, GRACE, HEART).
Does End-tidal carbon dioxide (EtCO2) level predict the risk of major cardiovascular events in patients with acute coronary syndrome?
Observational
Does End-tidal carbon dioxide (EtCO2) level predict the risk of major cardiovascular events in patients with acute coronary syndrome?
Answers are grounded in this paper and cite their sources.
EtCO2 levels, with an optimal cut-off of 30 mmHg, may serve as a useful non-invasive predictor for major cardiovascular events in patients with acute coronary syndrome.
OBJECTIVES: In this study, the capacity of End-tidal carbon dioxide (EtCO2) levels to predict the risk of major cardiovascular events (MACE) in patients diagnosed with acute coronary syndrome and the relationship between risk scoring systems (TIMI, GRACE, HEART) and EtCO2 values were examined. METHODS: EtCO2 values of the patients in the study were measured with a capnography device. Each patient's MACE status was recorded. RESULTS: < 0.00). (iii) The optimal EtCO2 cut-off point was 30 mmHg. This threshold value provided both sensitivity and specificity for the risk of MACE. (IV) Significant associations were found between EtCO2 and GRACE, TIMI and HEART scoring systems. EtCO2 values were significantly higher in the low-risk groups. (V) EtCO2 was significantly different in all three risk scoring systems in the non-MACE group, but only with the GRACE score in the MACE group. CONCLUSION: This study demonstrated that EtCO2 is a valuable indicator for predicting the risk of MACE in patients with ACS.
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Can et al. (2024) conducted an observational in Acute coronary syndrome. End-tidal carbon dioxide (EtCO2) measurement vs. Risk scoring systems (TIMI, GRACE, HEART) was evaluated on Major cardiovascular events (MACE). An end-tidal carbon dioxide cut-off of 30 mmHg provided sensitivity and specificity for predicting the risk of major cardiovascular events in patients with acute coronary syndrome.
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