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Thoracic veins, particularly pulmonary veins, are important arrhythmogenic foci that play a critical role in the pathophysiology of paroxysmal and permanent atrial fibrillation.
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Thoracic veins, especially pulmonary veins, are critical foci for atrial fibrillation genesis, and understanding their cellular electrophysiology may guide targeted ablation and pharmacological therapies.
The thoracic veins are important foci for the genesis of ectopic atrial tachycardia and play a critical role in the pathophysiology of paroxysmal and permanent atrial fibrillation. The pulmonary veins have the highest arrhythmogenic activity and other venous structures (eg, superior vena cava, coronary sinus and ligament of Marshall) have also been shown arrhythmogenic potential. Thoracic veins contain cardiomyocytes with distinct electrical activities and complex anatomical structures. This review summaries the current understanding of the basic and clinical electrophysiology of thoracic vein arrhythmias.
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Chen et al. (2007) conducted a review in Thoracic vein arrhythmias. Thoracic veins, particularly pulmonary veins, are important arrhythmogenic foci that play a critical role in the pathophysiology of paroxysmal and permanent atrial fibrillation.
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