Key result
Male sex, >12-hour delay, higher admission HR, anterior MI, and crackles predict post-AMI LV dysfunction.
Why the study?
LV dysfunction is a strong predictor of mortality in AMI, making the identification of patients at risk critical for early and optimal management.
Cohort (n=75)
No
In patients with acute myocardial infarction, male gender, delayed presentation, anterior infarction, higher heart rate, and clinical signs of heart failure predict the development of left ventricular dysfunction.
Left ventricular dysfunction (LV dysfunction) complicating myocardial infarction is common. Among patients with acute myocardial infarction (AMI), a strong relationship between the degree of LV dysfunction and mortality has been demonstrated. LV dysfunction is known to be a strong predictor of death in AMI with important implications for prognosis and treatment. The identification of patients at risk of developing LV dysfunction after AMI is relevant in the sense that it allows early and optimal management of heart failure for better quality of life. This prospective study of 75 cases of AMI complicated or not with LV dysfunction, hospitalized in the intensive care unit of the Mohammed V military training hospital in Rabat, covering a period of 6 months, aims to determine the predictive factors of LV dysfunction after an AMI in a Moroccan population group. It was found that male gender, delay in management, heart rate on admission, electrical territory, and the existence of crackling sounds on lungs at admission are predisposing factors for left ventricular systolic dysfunction. However, dyslipidemia is a protective factor. Early management of these patients would reduce the risk of reinfarction and death.
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Nikiema et al. (2021) conducted a cohort in Acute Myocardial Infarction (AMI) (n=75). Risk factors for LV dysfunction vs. Patients without LV dysfunction (LVEF > 40%) was evaluated on Left ventricular dysfunction (LVEF ≤ 40%). Male gender, delay in management >12 hours, higher admission heart rate, anterior infarction territory, and crackling lung sounds were significant predisposing factors for left ventricular systolic dysfunction after acute myocardial infarction.
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