Population
Sera from normal plasma, patients with Christmas deficiency, factor VII-deficient sera, and patients treated…
Design
Preclinical
Authors
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May explain heparin resistance after thrombosis; hypothesis-generating for serum factors, requiring human validation before clinical consideration.
The study identifies an antiheparin agent in serum that is not reduced by phenindione, suggesting a potential mechanism for hypercoagulability and heparin resistance in recent thrombosis.
L. Poller (1960) studied this question.
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