Cytoplasmic fluctuations generated by actomyosin networks enhance the transport efficiency of dynein molecular motors.
force measurements of dynein suggest that the asymmetric force response to random forces, generated by fluctuating dynamics of actomyosin networks, may contribute to acceleration. This study provides insights into a regulatory mechanism of molecular motors within fluctuating cytoplasm, harnessing cytoplasmic fluctuations to enhance transport efficiency in a highly crowded environment.
Torisawa et al. (Thu,) studied this question.
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