Key result
Post-infarction inflammation acts as a double-edged sword, being essential for the myocardial healing process but promoting adverse left ventricular remodeling when excessive.
Controlling excessive post-infarction inflammation represents an important potential therapeutic target for preventing pathological left ventricular remodeling and heart failure.
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Supports selective post-MI inflammation modulation; leaves open optimal agents and timing for trials.
Toshihisa Anzai (2013) conducted a review in Myocardial infarction and left ventricular remodeling. Post-infarction inflammation acts as a double-edged sword, being essential for the myocardial healing process but promoting adverse left ventricular remodeling when excessive.
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