Key result
Theoretical models of the dorsal mesocardium and ventricular myocardium demonstrated that two mechanically based hypotheses for the bending component of cardiac looping are consistent with known data.
Theoretical biomechanical models demonstrate that tension in the dorsal mesocardium or actin macrofilament contraction are feasible mechanisms for embryonic cardiac looping.
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Theoretical support for mechanical drivers of cardiac looping; leaves open in vivo validation and relevance to congenital defects.
Taber et al. (1995) studied Embryonic cardiac looping. Theoretical models of cardiac looping was evaluated on Feasibility of mechanically based hypotheses for the bending component of cardiac looping. Theoretical models of the dorsal mesocardium and ventricular myocardium demonstrated that two mechanically based hypotheses for the bending component of cardiac looping are consistent with known data.
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