Population
Rabbit hearts (n=14 for junctional rhythm origin study)
Design
Preclinical
Authors
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Supports connexin heterogeneity as basis for dual AV nodal pathways; hypothesis-generating for human AVNRT mechanisms.
Heterogeneous expression of connexin isoforms and uniform neurofilament expression in the AV nodal posterior extensions provide an anatomical and molecular basis for dual-pathway electrophysiology and AV nodal reentrant tachycardia.
Efimov et al. (2004) studied this question.
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