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October 1, 1993AJP Heart and Circulatory Physiology223 citations

Is rapid rise in vascular conductance at onset of dynamic exercise due to muscle pump?

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DSDon D. SheriffLRL. B. RowellASA. M. Scher

Key Points

  • This research aims to determine if rapid increases in vascular conductance at exercise onset are caused by the muscle pump.
  • Measured arterial and central venous pressure in nine awake dogs.

Structured PICO

Is the rapid rise in vascular conductance at the onset of dynamic exercise due to the muscle pump in awake dogs?

P
Population
9 awake dogs (8 with atrioventricular block, pacemakers, and ascending aortic flow probes; 1 with only an iliac artery probe)
I
Intervention
Dynamic exercise (0, 10%, and 20% grade at 4 mph) with and without autonomic blockade (hexamethonium)
C
Comparator
Pre-exercise baseline and varying exercise conditions
O
Outcome
Changes in vascular conductance, arterial pressure, and central venous pressure at the onset of exercisesurrogate

The rapid increase in vascular conductance at the onset of dynamic exercise is likely driven by the muscle pump rather than neural, metabolic, myogenic, or hydrostatic influences.

Abstract

We tested the hypothesis that rapid increases in muscle blood flow and vascular conductance (C) at onset of dynamic exercise are caused by the muscle pump. We measured arterial (AP) and central venous pressure (CVP) in nine awake dogs, eight with atrioventricular block, pacemakers, and ascending aortic flow probes for control of cardiac output (CO) (2 also had terminal aortic flow probes). One dog had only an iliac artery probe. At exercise onset (0 and 10% grade, 4 mph) C and CVP rose to early plateaus, and AP reached a nadir, all in 2-5 s. At 20% grade and 4 mph, C increased continuously after its initial sudden rise. Timing and magnitude of initial change in conductance (delta C) were independent of CO, AP, work rate (change in grade at constant speed), or autonomic function (blocked by hexamethonium). Speed of initial delta C and its independence from work rate and blood flow ruled out metabolic vasodilation as its cause; insensitivity to AP and autonomic blockade ruled out myogenic relaxation and sympathetic vasodilation as causes of sudden delta C. Sensitivity to contraction frequency (not work per se) implicates the muscle pump. When reflexes were blocked, a large secondary rise in C, presumably caused by metabolic vasodilation, began after 10 s of mild exercise. When reflexes were intact in mild exercise, C was lowered below its initial plateau by sympathetic vasoconstriction, which partially raised AP from its nadir toward its preexercise level. Our conclusion is that dynamic exercise has a large rapid effect on C that is not explained by known neural, metabolic, myogenic, or hydrostatic influences.(ABSTRACT TRUNCATED AT 250 WORDS)

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Cite This Study

Sheriff et al. (1993) studied this question.

synapsesocial.com/papers/6a20357760a84f4b7dd8dfe8https://doi.org/10.1152/ajpheart.1993.265.4.h1227
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