Key result
The I(NCX) current ensures intracellular Ca2+ homeostasis during early embryonic stages, but its density significantly decreases by the fetal stage while remaining regulated by Na+.
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No immediate clinical implications; leaves open NCX's role in fetal and adult Ca2+ dysregulation.
Reppel et al. (2007) studied this question. The I(NCX) current ensures intracellular Ca2+ homeostasis during early embryonic stages, but its density significantly decreases by the fetal stage while remaining regulated by Na+.
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