Key result
Subarachnoid hemorrhage frequently causes secondary cardiovascular complications, including electrocardiographic changes, regional wall motion abnormalities, neurogenic stunned myocardium, and elevated cardiac enzymes.
Subarachnoid hemorrhage frequently causes reversible cardiovascular complications such as neurogenic stunned myocardium and ECG changes due to sympathetic overactivity, which must be differentiated from primary acute coronary syndromes.
Highlights need for cardiac monitoring to differentiate neurogenic injury from ACS in SAH; leaves open prospective management trials.
Subarachnoid haemorrhage (SAH) is one of the devastating conditions, especially the aneurysmal bleed which has high mortality as well as morbidity. The mortality and morbidity caused by SAH have been recognised to be caused by both neurological and systemic causes. The alterations in systemic and other organ damage could cause death in up to 40% of SAH patients. Among the systemic manifestations, cardiovascular and respiratory complications increase hospitalisation and worsen the outcome. The main pathophysiological mechanism is the increased sympathetic activation causing myocardial necrosis. Various cardiovascular manifestations range from electrocardiogram changes to myocardial ischaemia, cardiac failure and arrhythmias. This review deals with the cardiac manifestations in SAH patients.
No takes yet. Share an insight, caveat, or question.
Manikandan Sethuraman (2017) conducted a review in Subarachnoid haemorrhage. Subarachnoid haemorrhage was evaluated. Subarachnoid hemorrhage frequently causes secondary cardiovascular complications, including electrocardiographic changes, regional wall motion abnormalities, neurogenic stunned myocardium, and elevated cardiac enzymes.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: