Key result
The combination of PKCβI and PKCβII inhibited BNaC2 activity, an effect that was abolished by a 5-fold excess of a PKCε and PKCζ combination.
Variable expression and functional antagonism of PKC isotypes regulate BNaC2 activity in glioma cells.
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Isoform antagonism on BNaC2 in glioma cells is hypothesis-generating; leaves open any therapeutic relevance in patients.
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Berdiev et al. (2002) studied Glioma. PKC isoforms was evaluated on BNaC2 activity. The combination of PKCβI and PKCβII inhibited BNaC2 activity, an effect that was abolished by a 5-fold excess of a PKCε and PKCζ combination.
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