Key result
Pharmacological regulation of macrophage polarization to control the M2/M1 ratio may help resolve inflammation, promote wound healing, and prevent cardiac fibrosis after myocardial infarction.
Population
Myocardial infarction
Design
Review
Authors
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May support macrophage-targeted therapies to limit post-MI fibrosis; hypothesis-generating and requires prospective trials.
This review highlights the therapeutic potential of targeting macrophage polarization to promote wound healing and prevent cardiac fibrosis after myocardial infarction.
Cheng et al. (2017) conducted a review in Myocardial infarction. Pharmacological regulators of macrophage polarization was evaluated. Pharmacological regulation of macrophage polarization to control the M2/M1 ratio may help resolve inflammation, promote wound healing, and prevent cardiac fibrosis after myocardial infarction.
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