Key result
Exercise modalities show potential for myocardial preservation by regulating cardioprotective heat shock proteins.
Why the study?
To systematically review advances on how heat shock proteins exert cardioprotective effects through multiple molecular mechanisms and examine how different exercise modalities achieve myocardial preservation by regulating heat shock protein expression.
Design
Systematic review
Authors
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Reinforces HSPs as exercise-inducible cardioprotective mediators; leaves open whether tailored regimens improve clinical outcomes.
Heat shock proteins play a central role in cardioprotection through multiple molecular mechanisms, and exercise serves as a potent physiological stressor to upregulate HSPs for non-pharmacological cardiovascular intervention.
ZHOU et al. (2026) conducted a review in Cardiovascular diseases and myocardial ischemia/reperfusion injury. Exercise modalities and Heat Shock Proteins (HSPs) regulation was evaluated. Heat shock proteins exert cardioprotective effects through multiple molecular mechanisms, and exercise modalities show potential in achieving myocardial preservation by regulating HSP expression.